New findings show that removing Centaurin-1, a protein elevated in Alzheimers disease, significantly reduces inflammation, plaque buildup, and cognitive deficits in a well-established mouse model. Eliminating this protein normalized several brain pathways, protected neural connections in the hippocampus, and improved spatial learning.
New findings show that removing Centaurin-1, a protein elevated in Alzheimers disease, significantly reduces inflammation, plaque buildup, and cognitive deficits in a well-established mouse model. Eliminating this protein normalized several brain pathways, protected neural connections in the hippocampus, and improved spatial learning.
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